Glia

We explore ways to manipulate glia to offer greater neuroprotection for retinal ganglion cells during glaucoma.

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Gangliosides

We endeavor to determine the contribution of gangliosides to glaucoma pathogenesis. The ganglioside GM1 undergoes changes in quantity and distribution during glaucoma.

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Mitochondria

We work to establish whether mitochondrial dysfunction contributes to glaucoma development. Mitochondria provide energy to all cells; we have observed ways in which energy management in the optic nerve is not normal in our glaucoma model.

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